ref for 1918 flu CFP

vignettes/brauer-ms.Rnw

@@ -2187,7 +2187,7 @@ legend("topleft", bty="n", lwd=c(2,lwd,lwd*0.60,lwd*0.6),
       eventually die if everyone in the city were infected, \ie the
       product of the population size of Philadelphia in 1918
       (\Sexpr{prettyNum(philapop1918, big.mark=",")}) and an assumed
-      CFP of $0.025$\needref.
+      CFP of $\Sexpr{philacfp}$ \cite{TaubMore06}.
       %%
       The fitted trajectory and confidence band are shown in
       \cref{fig:phila}.  See \cref{sec:phila}.}

vignettes/brauer-ms.bib

@@ -705,3 +705,16 @@ @Manual{mcstate
   note = {R package version 0.9.20},
   url = {https://github.com/mrc-ide/mcstate},
 }
+
+@article{TaubMore06,
+   Author = {Taubenberger, J. K. and Morens, D. M.},
+   Title = {1918 influenza: the mother of all pandemics},
+   Journal = {Emerging Infectious Diseases},
+   Volume = {12},
+   Number = {1},
+   Pages = {15-22},
+   Abstract = {The "Spanish" influenza pandemic of 1918-1919, which caused 50 million deaths worldwide, remains an ominous warning to public health. Many questions about its origins, its unusual epiclemiologic features, and the basis of its pathogenicity remain unanswered. The public health implications of the pandemic therefore remain in doubt even as we now grapple with the feared emergence of a pandemic caused by H5N1 or other virus. However, new information about the 1918 virus is emerging, for example, sequencing of the entire genome from archival autopsy tissues. But, the viral genome alone is unlikely to provide answers to some critical questions. Understanding the 1918 pandemic and its implications for future pandemics requires careful experimentation and in-depth historical analysis.},
+   Keywords = {Spanish influenza; virus hemagglutinin; a viruses; receptor; gene;
+mortality; evolution; sequences; strains; segment},
+   Year = {2006} }
+